Passive exercises for children with developmental delay or brain injury

Passive exercises for children with developmental delay or brain injury

passive exercises

Autism, also known as autism spectrum disorder (ASD), is a developmental disorder that affects communication and social interaction. The severity of ASD can vary widely, and different individuals with ASD may experience different symptoms.

First month

  1. rocking, listening to fairy tales, the tale should be about the child.
  2. swinging: for 20 minutes, during which we rhymes
  3. rotation as long as they can stand: child stands with his back to the parent, holds it under his armpits and the parent rotate the child
  4. rolling around the longitudinal axis; you can carefully wrap the child in a blanket and spin it out by holding one end of the blanket. Older kids should roll about 40m
  5. Hip rotation: hands in baby posture during lying on the back, feet on soles, knees-head turning in opposite directions
  6. Throwing into a pillow from different heights, if the child is big, just push into the pillow 20x. A large bean bag is the best for this
  7. Jumping on a trampoline: if the child is small just sit the child in front of your leg and you jump, the kid bounces up and down
  8. Vigorous massaging of fingertips and nail beds on hands and feet, there are a lot of nerve endings there, this is a powerful stimulation for the child

Second month

  1. Rocking, and listening to fairy tales or music
  2. Swinging: rotate in the swing and let it spin 5-6x
  3. Rotate upside down, too, holding the hips of the child. In the case of a bigger kid use a swivel chair and lie the kid upside down.
  4. Back rotation, holding the back of the head, walking around the parent
  5. Hip rotation, with legs stretched.
  6. Throwing into a pillow while lying down with chin pressed against chest (hold your favorite plush)
  7. Jumping on trampoline

In the case of an autistic child: if there is a problem with touch: the sense of touch may be underactive or the child cannot be touched:
• 20x20cm piece of velvet, silk, linen, terrycloth, or other material

• caress the child with a different substance every day; later the child should choose which one he wants

Third month

  1. Rocking, listening to fairy tales, music, over 5 years old content telling can also be requested
  2. Swinging, balance swinging.
  3. Flying: child lying on his stomach dad is standing and he starts spinning with the child in a circle or use swivel chair
  4. Rotation: lying down on the back, hands in baby pose, legs pulled up, hip rotation, head against
  5. Standing, head nodding forward 10x , back 10x
  6. Unfolding from the packaging; the child lies on his back, wrapp up his hand or feet, left hand, right hand, left foot, right foot, head 10x
  7. Closing fingers one by one, with rhymes, in the rhyme should include the name of the finger; we can hold the fingers or we move them
  8. Jumping on trampoline
Resources
  • m B-R, SeoH-S, Ku J-M, et al. Silibinininhibits the production of pro-inflammatory cytokines through inhibition of NF-κB signaling pathway in HMC-1 human mast-cells. Inflammation. Research. 2013;62(11):941-950. doi:10.1007/s00011-013-0640-1.
  • ChapowalA. PetasitesStudy Group. Randomisedcontrolled trial of butterbur and cetirizine for treating seasonal allergic rhinitis. BMJ 2002;324:144-6.
  • Hayes, N. A. and Foreman, J. C. The activity of compounds extracted from feverfew on histamine release from rat mast-cells. J Pharm Pharmacol1987;39(6):466-470
  • Hsieh et al. Baicalein inhibits IL-1ß- and TNF-a-induced inflammatory cytokine production from human mast-cells via regulation of the NF-?B pathway. ClinMolAllergy. 5: 5. 2007.
  • TheoharidesTC, Patra P, Boucher W, et al. Chondroitin sulphateinhibits connective tissue mast-cells. British Journal of Pharmacology. 2000;131(6):1039-1049. doi:10.1038/ sj.bjp.0703672.
  • Ro JY, Lee BC, Kim JY, et al. Inhibitory mechanism of aloe single component (alprogen) on mediator release in guinea pig lung mast-cells activated with specific antigenantibodyreactions. J PharmacolExpTher. 2000;292:114–121. 73.
  • https://www.ncbi.nlm.nih.gov/pubmed/24477254
  • https://www.ncbi.nlm.nih.gov/pubmed/28458279
  • https://www.ncbi.nlm.nih.gov/pubmed/9421440
  • https://www.ncbi.nlm.nih.gov/pubmed/10344773
  • https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4315779/
Delacato method: with movement for intellectual development

Delacato method: with movement for intellectual development

Carl-Henry Delacato method

According to the theory developed by Philadelphia neurologist Carl Henry Delacato and his team, there is a close connection between movement and the development of the nervous system, and intellectual development. So if the child does not have a way to practice some form of movement, it must be replaced later for proper brain development.

According to the theory, individual development follows the path of stock development, so the child copies the development process that took place when he became a human in his first years.

There is a close connection between movement and the maturation of the nervous system. If the child did not have time to practice a form of movement – either because he missed it or because the next step followed too soon, for example he started walking very early and therefore did not climb for long enough – this affects later development and even causes learning difficulties and can also cause behavior.

According to the method, the child relives an earlier period of life through movement, thus bringing in your backlog.

Delacato method can be used for intellectual development?

  • Delayed/defective/stuttering speech development
  • Speech error
  • Dyslexia/dysgraphia
  • Attention deficit disorder
  • Mild intellectual disability
  • Clumsy movement
  • Poor orientation skills
  • Integration problems

How does the treatment take place?

According to Delacato, elementary movements – crawling, climbing, walking, swimming – reveal the development of each area of ​​the nervous system. After a condition assessment, the child is given a set of 8-10 exercises, which must be repeated at home for 20-30 minutes every day. You only have to appear at one control examination a month, when you will receive a new set of exercises taking into account your progress.

The tasks develop balance, spatial perception and fine movement.

It is becoming more and more common for specialists who mainly deal with large groups to visit kindergartens as well. In such cases, 90-minute classes are held for groups of 4-5 people.

Children and young people between the ages of 5 and 16 can take part in the therapy. Since the goal of the development is to practice the missed forms of movement,teenagers do the same exercises that preschoolers do: crawling, climbing, balancing, allowing their brains and nervous systems to acquire skills that were missed in the first period.

Let's have some exercise

Check out some brain developing exercises in the related articles:

Passive exercises for children with developmental delay or brain injury

Exercises for brain and intellectual development Part 1

 

Movement developmental milestones: What should you pay attention to?

Movement developmental milestones: What should you pay attention to?

development milestones

Children grow and develop at their own pace. This is a fact that a parent must be able to accept, just like the fact that their child may start walking or talking later. If your child does not reach certain developmental milestones, it is recommended to visit a specialist. Which milestones should you focus on?

There are children who, in terms of development, show almost a textbook example and reach each milestone on a daily basis. Then there are children who surpass their peers in everything, and there are those who develop at a slower pace , but still within the tolerance limit. When should you start worrying?

At the age of 0 - 4 months

Babies grow very quickly in the first months after birth, so parents can witness changes on a daily basis. A healthy newborn gains 140-200 grams in the first week and grows 1-2.5 cm per month. Children literally grow before our eyes and learn new skills at the same time. They must reach a certain level of development in the first four months of their lives . If you notice any of the following symptoms during this period, see your pediatrician as soon as possible, as they may indicate a developmental or other abnormality:

  • squeals
  • does not pay attention to loud noises
  • he doesn’t look at his hands
  • does not follow moving objects with his eyes
  • does not grab objects
  • he does not smile or laugh at the people around him
  • he can’t keep his head
  • not goofy and/or imitate sounds
  • does not put objects in his mouth
  • does not strain its paws when they touch a solid surface

At the age of 5 – 7 months

As the baby grows, its abilities and skills also develop. The five- to seven-month-old baby smiles, laughs, is excited . He grabs different objects, tastes them, snorts and makes noise. His world is expanding and he is discovering more and more of it every day. As with the previous one, there are defining milestones in this period , so be aware if you experience the following symptoms:

  • his posture is stiff, his muscles tense
  • his muscles are relaxed, with a rag doll posture
  • he does not hold his head, his head leans back even in a sitting position
  • he only reaches for the objects with one hand
  • rejects babysitting
  • it is not attached to the person taking care of it
  • he is sensitive to light, his eyes water
  • he finds it difficult to put his hand in his mouth
  • it does not turn over in the fifth month
  • he can’t even sit with help until the sixth month
  • you don’t laugh or make a sound until the sixth month

At the age of 8 – 12 months

When they light the first candle on the birthday cake, they most likely already have a ready-made personality at home. The past months have brought physical, mental, social and emotional changes in the baby’s life, and in terms of development, further milestones lie ahead. Around 75% of children achieve these in a certain period. Watch for the following symptoms:

  • does not climb or does not climb regularly, e.g. pulls one side
  • unable to stand even with support
  • does not search for objects hidden in plain sight
  • he hasn’t said the first word yet
  • does not gesticulate, e.g. he does not shake his head at the answer “no”.
  • does not point to pictures or objects

Up to 2 years old

Intensive development also takes place during childhood. At the age of two, the majority of children are walking, talking, jumping, running … a little one is simply everywhere. During this very active period, many parents notice when something is wrong with their child. Why do not you speak? Why doesn’t he come? Why don’t you play the games? If you notice any of the following symptoms, do not hesitate and notify your child’s doctor:

  • knows and uses less than 15 words
  • does not use two-word sentences
  • does not imitate words, sounds or movements
  • unable to follow simple instructions (not to be confused with disobedience)
  • cannot push wheeled toys
  • It does not go until the age of 18 months
  • even if you walk, you don’t use all parts of your foot (ie from toe to heel)

At the age of 3

The time flew by in a flash and it has now dwindled to three years. If your child has a developmental disorder , you’ve probably already discovered it, but there are still symptoms that cause concern:

  • often falls and cannot climb stairs
  • drools continuously and/or his speech is unintelligible
  • cannot use more than 4 cubes when building a tower
  • cannot manipulate small objects
  • cannot draw a circle
  • unable to communicate in short sentences
  • he is not interested in group play, he does not participate in it
  • does not understand simple instructions
  • no longer interested in children
  • does not maintain eye contact
  • shows little interest in games

A parent knows their child best. He is the best able to assess his development and follow the progression of his behavior and skills. We always emphasize that comparison with peers is inappropriate because children develop at their own pace, but this does not mean that the child should not be monitored. If you feel that everything is not right, or if you have noticed one of the symptoms mentioned above , contact a specialist who will order the necessary tests.

Resources
  • m B-R, SeoH-S, Ku J-M, et al. Silibinininhibits the production of pro-inflammatory cytokines through inhibition of NF-κB signaling pathway in HMC-1 human mast-cells. Inflammation. Research. 2013;62(11):941-950. doi:10.1007/s00011-013-0640-1.
  • ChapowalA. PetasitesStudy Group. Randomisedcontrolled trial of butterbur and cetirizine for treating seasonal allergic rhinitis. BMJ 2002;324:144-6.
  • Hayes, N. A. and Foreman, J. C. The activity of compounds extracted from feverfew on histamine release from rat mast-cells. J Pharm Pharmacol1987;39(6):466-470
  • Hsieh et al. Baicalein inhibits IL-1ß- and TNF-a-induced inflammatory cytokine production from human mast-cells via regulation of the NF-?B pathway. ClinMolAllergy. 5: 5. 2007.
  • TheoharidesTC, Patra P, Boucher W, et al. Chondroitin sulphateinhibits connective tissue mast-cells. British Journal of Pharmacology. 2000;131(6):1039-1049. doi:10.1038/ sj.bjp.0703672.
  • Ro JY, Lee BC, Kim JY, et al. Inhibitory mechanism of aloe single component (alprogen) on mediator release in guinea pig lung mast-cells activated with specific antigenantibodyreactions. J PharmacolExpTher. 2000;292:114–121. 73.
  • https://www.ncbi.nlm.nih.gov/pubmed/24477254
  • https://www.ncbi.nlm.nih.gov/pubmed/28458279
  • https://www.ncbi.nlm.nih.gov/pubmed/9421440
  • https://www.ncbi.nlm.nih.gov/pubmed/10344773
  • https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4315779/
Brain plasticity exercises

Brain plasticity exercises

Brain neuroplasticity in asd

Neuroplasticity refers to the brain’s ability to reorganize itself by forming new connections between brain cells (neurons) and by altering the strength of existing connections. This process allows the brain to adapt and learn new skills, as well as to compensate for injuries or diseases.

How to improve the brain neuroplasticity?

Several studies have demonstrated that brain plasticity improves brain recovery neurologists often use brain plasticity to describe rehabilitation after injury. Adult brains are not hard-wired; it is crucial to keep this in mind in the brain. Many wirings are created with a fixed neural circuit. The neural connections aren’t what most people think they are. There is scientific evidence that cortical neurons don’t die as we age. Injuries and training lead to subcortical rewiring in the brain.

Brain Plasticity: Exercises part 1

Research has shown that neuroplasticity can be enhanced through certain activities and experiences. Many scientists believe that humans need to exercise their brains for a long time during childhood, the brain stops developing, so we lose neurons every year until we reach adulthood
modern research indicates that the brain is still capable of developing.
Throughout our lives, we form neural connections based on our experiences.

To stay healthy, you must exercise both your brain and your body. We lose neurons every year until age because many scientists believe the brain stops developing in childhood. Researchers have found, however, that our brains continue to establish neural connections throughout our lives.
addition,
Everyone can improve neuroplasticity by doing exercises that can make the brain even stronger despite the factors leading to brain damage, like environmental stress, drugs, and aging. Like doing aerobic exercises for your heart, it’s just as important to condition your brain by exercising regularly.

Rules for Brain Plasticity Workouts

Before you get started exercising the brain, it is important to make sure you are aware of the cautions and rules in order to ensure that you are getting the best results and get the most out of it. Neurons that are not stimulated well will die so all of your attempts to reconnect will be useless. Below are the rules:

Practice regularly

Neurons that are not stimulated enough usually fail to reconnect. This is especially true if somebody has been suffered from brain injuries. It is crucial to start doing neuroplasticity exercises even before your neurons start to die. If you practice regularly, not only will your neural connections flourish but your neurons will also be regenerated according to many studies.

Learn new skills

Learning new skills, such as a new language or instrument, can stimulate the formation of new connections in the brain and enhance neuroplasticity.

Exercises need to be balanced.

It is important to take note that repeating brain exercises for countless of times can lead to the overstimulation of the brain cells in the targeted areas.

Practice every day.

Exercise regularly. It is worth constantly stimulating the brain with varied tasks.

What activities increase neuroplasticity?

  • 3D adventure games appeared to improve memory, problem-solving, and scene recognition.
  • Puzzles improve problem-solving skills, brain connections, and spatial prediction.
  • Rhythmic games, such as dance or exercise video games, can improve visuospatial memory and attention.
  • Learn a new language. A second (or third) language might improve your career prospects, or you want to learn it for fun. Either way, you’re doing your brain a big favor. There is a lot of evidence that learning a new language improves cognitive function. Increase the grayness…In a 2012 study, researchers studied ten English-speaking exchange students studying German in Switzerland. After five months of intensive language training, their German language skills increased – and so did the density of gray matter in their brains. Your brain has many vital areas of gray matter, including areas related to language, attention, memory, emotions motor skills. Increasing gray matter density can improve your function in these areas, especially with age. Bilingualism is thought to offer protection against cognitive decline. Language learning at any stage of life can slow future decline with age and the symptoms of dementia. Learning a new language increases gray matter density and neuroplasticity.
  • Music has many benefits for the brain. It can help improve: your mood, and your ability to learn and remember new information; concentrate and concentrate; music therapy also appears to help slow cognitive decline in older adults. According to a 2017 study, music, especially when combined with dance, art, play, and exercise, helps promote neuroplasticity. It can improve movement and coordination and can help strengthen memory. But it simply won’t help prevent further cognitive decline. It can also help relieve emotional stress and improve quality of life. According to a 2015 review, musical training is also useful as neuroplasticity training. Learning to play music in childhood may help protect against age-related cognitive decline and improve cognitive performance in older adulthood. A reliable source also suggests that musicians often have: better auditory and visual perception, better concentration, better memory better motor coordination. It’s never too late to learn an instrument.
  • Most people know that exercise offers many physical benefits but physical activity also strengthens the brain. Exercise – especially aerobic exercise – can improve cognitive skills such as learning and memory. According to a 2018 literature review, exercise helps improve fine motor skills and brain connections and may protect against cognitive decline. Neuroplasticity exercise helps promote blood flow and cell growth in the brain. You’ll likely see social benefits if you train with someone else or in a larger group. Social connections help improve quality of life and emotional well-being, so regular interaction with others can be another great way to improve brain health and reduce symptoms of anxiety and depression. Exercise recommendations may vary based on health and ability, but it’s a good idea to get at least some exercise daily.
  • Making art can help you see the world in a new and unique way. You can use art to sort out and express emotions, share personal experiences, or gain deeper insights.
  • Get enough sleep: Sleep is important for brain health because it allows the brain to rest and repair itself. Adequate sleep can improve brain function and increase neuroplasticity.
  • Healthy diet: A diet rich in vegetables, fruits, and other nutrients has been shown to support brain health and increase neuroplasticity.
  • Practice meditation: Meditation and mindfulness practices have been shown to improve brain function and increase neuroplasticity.
  • Stay mentally active: Engaging in mentally challenging activities like puzzles and games can help keep your brain active and stimulate the growth of new brain cells.
  • Be Socially Active: Connecting with others and maintaining strong social ties has been shown to benefit brain health and increase neuroplasticity
Resources
  • m B-R, SeoH-S, Ku J-M, et al. Silibinininhibits the production of pro-inflammatory cytokines through inhibition of NF-κB signaling pathway in HMC-1 human mast-cells. Inflammation. Research. 2013;62(11):941-950. doi:10.1007/s00011-013-0640-1.
  • ChapowalA. PetasitesStudy Group. Randomisedcontrolled trial of butterbur and cetirizine for treating seasonal allergic rhinitis. BMJ 2002;324:144-6.
  • Hayes, N. A. and Foreman, J. C. The activity of compounds extracted from feverfew on histamine release from rat mast-cells. J Pharm Pharmacol1987;39(6):466-470
  • Hsieh et al. Baicalein inhibits IL-1ß- and TNF-a-induced inflammatory cytokine production from human mast-cells via regulation of the NF-?B pathway. ClinMolAllergy. 5: 5. 2007.
  • TheoharidesTC, Patra P, Boucher W, et al. Chondroitin sulphateinhibits connective tissue mast-cells. British Journal of Pharmacology. 2000;131(6):1039-1049. doi:10.1038/ sj.bjp.0703672.
  • Ro JY, Lee BC, Kim JY, et al. Inhibitory mechanism of aloe single component (alprogen) on mediator release in guinea pig lung mast-cells activated with specific antigenantibodyreactions. J PharmacolExpTher. 2000;292:114–121. 73.
  • https://www.ncbi.nlm.nih.gov/pubmed/24477254
  • https://www.ncbi.nlm.nih.gov/pubmed/28458279
  • https://www.ncbi.nlm.nih.gov/pubmed/9421440
  • https://www.ncbi.nlm.nih.gov/pubmed/10344773
  • https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4315779/
Benefits of neuroplasticity

Benefits of neuroplasticity

Benefit of neuroplasticity

Neuroplasticity can be a powerful tool for restoring normal brain functions, as well as for treating a range of neurological and mental health issues. So neuroplasticity can be beneficial not only in the case of brain injury but autism as well.

Benefits of Neuroplasticity

One of the main benefits of neuroplasticity is its ability to help us recover from physical and mental injuries. For example, neuroplasticity has been used to help stroke victims regain lost motor and sensory functions. Neuroplasticity can also be used to treat neurodegenerative diseases like Parkinson’s, as well as mental illnesses such as depression and anxiety. Neuroplasticity can be used to rewire neural pathways in order to make them stronger, more efficient, and more resilient.

Neuroplasticity can also be used to improve cognitive abilities such as memory, attention, and learning. It has been used to help people with learning disabilities, including dyslexia, by allowing them to make better use of their existing neural pathways. By strengthening existing connections and creating new ones, neuroplasticity can help people learn new skills more quickly.

Neuroplasticity can be used to help us stay mentally fit and healthy throughout our lives. By stimulating our brains in different ways – for example, by learning new things or practicing mindfulness – we can keep our minds sharp and resilient. Neuroplasticity gives us the ability to adapt to changes in our environment or lifestyle, and this can help us stay mentally fit and healthy into old age.

The Limitations of Neuroplasticity

Neuroplasticity is an incredibly powerful and effective tool for healing the brain, but it does have its limitations. For one, neuroplasticity doesn’t work as quickly or efficiently as some people think. It’s important to remember that neuroplasticity can take time to fully manifest in the brain, and depending on the condition, it may not ever fully cure the issue at hand.

Additionally, neuroplasticity isn’t always a permanent fix. The brain is constantly changing and adapting, which means that neuroplasticity may be helpful in the short-term but not necessarily the long-term. It is also possible that changes made through neuroplasticity can become unstable over time, leading to the original problem returning.

Finally, neuroplasticity has its limits in terms of what it can treat. Neuroplasticity is largely focused on healing physical brain issues, such as certain types of brain injuries, rather than mental health issues like depression and anxiety.

Overall, neuroplasticity is a useful tool for healing the brain, but it is important to be aware of its limitations. With proper awareness and care, neuroplasticity can be used effectively to treat a variety of conditions.

Related articles:

Brain plasticity exercises Part1

Can the brain be cured? – What is neuroplasticity?

Resources
  • m B-R, SeoH-S, Ku J-M, et al. Silibinininhibits the production of pro-inflammatory cytokines through inhibition of NF-κB signaling pathway in HMC-1 human mast-cells. Inflammation. Research. 2013;62(11):941-950. doi:10.1007/s00011-013-0640-1.
  • ChapowalA. PetasitesStudy Group. Randomisedcontrolled trial of butterbur and cetirizine for treating seasonal allergic rhinitis. BMJ 2002;324:144-6.
  • Hayes, N. A. and Foreman, J. C. The activity of compounds extracted from feverfew on histamine release from rat mast-cells. J Pharm Pharmacol1987;39(6):466-470
  • Hsieh et al. Baicalein inhibits IL-1ß- and TNF-a-induced inflammatory cytokine production from human mast-cells via regulation of the NF-?B pathway. ClinMolAllergy. 5: 5. 2007.
  • TheoharidesTC, Patra P, Boucher W, et al. Chondroitin sulphateinhibits connective tissue mast-cells. British Journal of Pharmacology. 2000;131(6):1039-1049. doi:10.1038/ sj.bjp.0703672.
  • Ro JY, Lee BC, Kim JY, et al. Inhibitory mechanism of aloe single component (alprogen) on mediator release in guinea pig lung mast-cells activated with specific antigenantibodyreactions. J PharmacolExpTher. 2000;292:114–121. 73.
  • https://www.ncbi.nlm.nih.gov/pubmed/24477254
  • https://www.ncbi.nlm.nih.gov/pubmed/28458279
  • https://www.ncbi.nlm.nih.gov/pubmed/9421440
  • https://www.ncbi.nlm.nih.gov/pubmed/10344773
  • https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4315779/
Can the brain be cured? – What is neuroplasticity?

Can the brain be cured? – What is neuroplasticity?

what is neuroplasticity of the brain

Have you ever wondered if the brain can be “cured”? This is a question that has been debated for centuries, but recent research into the science of neuroplasticity may provide the answer. Neuroplasticity is the idea that the brain can change and adapt throughout our lives, and that these changes can be beneficial to our overall mental health and wellbeing. In this blog post, we will explore what neuroplasticity is, how it works, and whether it can be used to “cure” the brain.

The brain has the ability to change and adapt, a phenomenon known as neuroplasticity. Neuroplasticity refers to the brain’s ability to reorganize itself by forming new connections between brain cells (neurons) and by altering the strength of existing connections. This process allows the brain to adapt and learn new skills, as well as to compensate for injuries or diseases.

Electrical impulses travel through neurons to carry information

A neurotransmitter is released when the body receives it. A neurotransmitter is released when the body receives it. Released neurotransmitters bind to receptors through the synaptic cleft and create new synapses post-synaptic neurons fire electrical signals that cause the brain to fire as a result. The synapse can modify itself according to how strong it is. A neuron’s ability to alter its shape is called neuroplasticity. There is a reason why our brains can constantly learn new information even without us realizing it is a seemingly overburdened system.
Neuroplasticity refers to how a neuron’s brain-cell synapses grow or decrease in strength depending on the use or lack thereof.

Neuroplasticity, sometimes referred to as brain plasticity which is the capability of the brain to form new connections between neurons. Neuroplasticity is an adaptive feature of the brain that enables it to adjust its function in response to changes in the environment or within the body. It allows the brain to modify its structure and circuitry, responding to learning, experience, and injury.
The concept of neuroplasticity is not new; scientists have known about it since the late 19th century. However, it wasn’t until recently that neuroscientists began to understand the full extent of its power and potential.
Neuroplasticity occurs when the brain adapts to external stimuli or internal needs. It involves the modification of existing neural pathways and the formation of new ones. This process allows for a more efficient and effective information organization, leading to improved mental performance and the potential for improved memory, language, and cognition.
Neuroplasticity can occur at any age, and it plays a key role in memory, learning, and the ability to adapt to new situations. Research has also shown that neuroplasticity can be enhanced through certain activities, such as learning new skills, practicing meditation, and engaging in physical exercise.
While neuroplasticity offers the potential for the brain to adapt and regenerate, it is not always possible to fully “cure” certain brain conditions or diseases. In some cases, neuroplasticity may be able to help compensate for damage or dysfunction in the brain, but it is not always possible to fully restore lost function.

The Three Main Types of Neuroplasticity

There are three main types of neuroplasticity: Hebbian plasticity, activity-dependent plasticity, and homeostatic plasticity.

Hebbian plasticity is the type of neuroplasticity where neurons that fire together, wire together. This type of plasticity is based on the idea that when two neurons are activated simultaneously, their connection is strengthened. This is why it is so important to practice new skills or behaviors in order for them to become ingrained in your brain.

Activity-dependent plasticity occurs when neural connections are strengthened or weakened based on experience and activity. This type of plasticity occurs when an individual engages in activities that require concentration and focus. For example, if someone spends a great deal of time practicing a musical instrument, their neural connections associated with playing that instrument will become stronger over time.

Homeostatic plasticity is the type of neuroplasticity that helps regulate the firing rate of neurons. This type of plasticity allows the brain to adjust its responses to different stimuli by fine-tuning the connections between neurons. For example, when someone experiences chronic stress, their brains may increase their sensitivity to stressful stimuli in order to protect itself from further harm. Homeostatic plasticity helps the brain maintain a balance between different states.

How Does Neuroplasticity Work in the Brain?

The first step in the process is for the brain to recognize something new. This could be a new idea, concept, or experience. Once the brain is aware of this new stimulus, it begins to form pathways and connections between different parts of the brain.

It can form these connections in several different ways. For example, the brain may increase communication between two different areas or strengthen an existing connection. It can also weaken connections that are no longer needed or even create entirely new ones.

These changes don’t happen overnight, though. Neuroplasticity is a long-term process, so it takes time for the brain to adjust to the new stimulus and make these changes permanent. The end result is a more efficient and effective brain that can handle complex tasks with ease.

Ultimately, neuroplasticity is an amazing process that helps us learn and adapt to our environment. By understanding how it works, we can develop strategies and techniques to optimize our mental performance and ultimately get the most out of life.

In the case of autism, it is worth taking advantage of the possibility of neuroplasticity, as it can greatly contribute to the development of learning abilities, cognitive abilities, and memory. In our next article, we list tasks that promote neuroplasticity

Related articles:

Brain plasticity exercises Part1

Benefits of Neuroplasticity

Resources
  • m B-R, SeoH-S, Ku J-M, et al. Silibinininhibits the production of pro-inflammatory cytokines through inhibition of NF-κB signaling pathway in HMC-1 human mast-cells. Inflammation. Research. 2013;62(11):941-950. doi:10.1007/s00011-013-0640-1.
  • ChapowalA. PetasitesStudy Group. Randomisedcontrolled trial of butterbur and cetirizine for treating seasonal allergic rhinitis. BMJ 2002;324:144-6.
  • Hayes, N. A. and Foreman, J. C. The activity of compounds extracted from feverfew on histamine release from rat mast-cells. J Pharm Pharmacol1987;39(6):466-470
  • Hsieh et al. Baicalein inhibits IL-1ß- and TNF-a-induced inflammatory cytokine production from human mast-cells via regulation of the NF-?B pathway. ClinMolAllergy. 5: 5. 2007.
  • TheoharidesTC, Patra P, Boucher W, et al. Chondroitin sulphateinhibits connective tissue mast-cells. British Journal of Pharmacology. 2000;131(6):1039-1049. doi:10.1038/ sj.bjp.0703672.
  • Ro JY, Lee BC, Kim JY, et al. Inhibitory mechanism of aloe single component (alprogen) on mediator release in guinea pig lung mast-cells activated with specific antigenantibodyreactions. J PharmacolExpTher. 2000;292:114–121. 73.
  • https://www.ncbi.nlm.nih.gov/pubmed/24477254
  • https://www.ncbi.nlm.nih.gov/pubmed/28458279
  • https://www.ncbi.nlm.nih.gov/pubmed/9421440
  • https://www.ncbi.nlm.nih.gov/pubmed/10344773
  • https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4315779/
Oxidative Stress May Be Linked to Autism

Oxidative Stress May Be Linked to Autism

oxidative stress and autism

Research suggests that there may be a link between autism and oxidative stress. A recent study found that metabolic endophenotypes and genotypes associated with autism are associated with higher levels of oxidative stress in children with the disorder. This is an important discovery that could help us better understand the causes of autism and lead to more effective treatments for those affected by it.

One research utilized a longitudinal study design to explore the relationship between metabolic endophenotype, related genotypes, and oxidative stress in children with autism. Metabolic endophenotypes were measured via urine and plasma samples collected from participants at three different times throughout the study. Genotype data was also obtained and analyzed for any potential associations with the observed oxidative stress. In order to assess oxidative stress, specific biomarkers of oxidative damage were monitored over the course of the study.

In addition, demographic information was obtained from each participant, such as age, sex, and body mass index (BMI). This information was used to ensure that any potential associations between metabolic endophenotypes, genotypes, and oxidative stress did not occur due to confounding factors. Statistical analyses were then performed to determine the strength of these associations.

Result

The study on metabolic endophenotype and related genotypes in children with autism found a strong correlation between oxidative stress and autistic characteristics. The researchers studied a total of 117 children who had been diagnosed with Autism Spectrum Disorder (ASD) and 88 typically developing children.

Blood samples were taken from all the participants, and markers of oxidative stress were measured. It was found that there were higher levels of markers of oxidative stress in the autistic group than in the typically developing group. Additionally, researchers identified an association between certain genotypes and oxidative stress in the autistic group.

Interestingly, the study revealed that the association between certain genetic markers and oxidative stress was strongest for autistic symptoms related to cognitive impairment, communication deficits, and repetitive behaviors. This suggests that oxidative stress may play a role in the severity of these symptoms in people with autism.

Conclusion

The results of this study suggest a possible link between metabolic endophenotype, related genotypes, and oxidative stress in children with autism. This link is significant because oxidative stress has been linked to many different health issues, such as cardiovascular disease, diabetes, cancer, and neurodegenerative diseases. Additionally, oxidative stress may also play a role in autism by affecting development and cognition.

Although the study found an association between metabolic endophenotype, related genotypes, and oxidative stress in children with autism, further research is needed to confirm the findings. Additionally, more research is needed to determine the mechanisms by which these factors might be associated with oxidative stress. It is also important to consider the potential confounding effects of other variables, such as diet, lifestyle, and environmental exposures.

Overall, the findings of this study provide new insight into the potential role of metabolic endophenotype and related genotypes in autism and suggest that oxidative stress may be an important factor in the development of the condition. Researchers stated: “We propose that an increased vulnerability to oxidative stress (endogenous or environmental) may contribute to the development and clinical manifestations of autism.” (1)

Resources

https://pubmed.ncbi.nlm.nih.gov/16917939/

Language Processing Disorder: When Words Don’t Make Sense

Language Processing Disorder: When Words Don’t Make Sense

LPD

Language Processing Disorder (LPD) is a communication disorder that affects a person’s ability to comprehend and use language. It is also known as Central Auditory Processing Disorder (CAPD) and affects both children and adults. People with LPD have difficulty understanding and producing language. They often find it difficult to follow instructions, interpret what others are saying, or express themselves using language. In this blog post, we will explore the symptoms, causes, and treatment of Language Processing Disorder.

What is Language Processing Disorder?

Language Processing Disorder (LPD) is a communication disorder characterized by difficulty understanding and using spoken and written language. It can affect all areas of communication including listening, speaking, reading, and writing. People with LPD have trouble comprehending spoken language and interpreting what is said or written. They may have difficulty formulating responses, organizing their thoughts, and understanding figurative language such as idioms or metaphors. This disorder is often seen in individuals with autism or other developmental disorders. In general, people with LPD are very literal and interpret language in its most basic form.

Symptoms of LPD

Symptoms of LPD may include difficulty understanding spoken language, difficulty expressing oneself through spoken or written language, and difficulty with reading comprehension. Some individuals with LPD may also have difficulty with language-related tasks, such as following directions or answering questions.

LPD is often associated with other conditions, such as autism spectrum disorder (ASD) or attention deficit hyperactivity disorder (ADHD). It is important to note that LPD is not a stand-alone diagnosis, and a thorough evaluation by a healthcare professional is typically needed to determine whether an individual has LPD and to develop an appropriate treatment plan. Treatment may include speech therapy or other interventions to improve language skills and communication.

Causes of LPD

The exact cause of Language Processing Disorder (LPD) is unknown. It is believed to be the result of a combination of genetic and environmental factors. Research suggests that LPD may be hereditary, as it can run in families. Additionally, certain types of hearing problems can make it more difficult for individuals to process language, which may lead to LPD.

Some studies have found an association between autism spectrum disorder (ASD) and language processing difficulties. People with ASD may have difficulty understanding language in all its forms, including non-verbal communication, such as body language and facial expressions. They may also have difficulty interpreting subtle nuances in language, such as sarcasm or jokes. This can make it difficult for them to understand conversations and learn new words.

Overall, the exact cause of LPD remains unknown, but it is likely due to a combination of genetic, environmental, and/or neurological factors.

Diagnosing LPD

Language Processing Disorder (LPD) can be difficult to diagnose. It is often misdiagnosed as a learning disability or Attention Deficit Hyperactivity Disorder (ADHD). In order to accurately diagnose LPD, a comprehensive assessment must be conducted by an experienced professional.

The assessment should include assessments of language comprehension, expression, and cognitive abilities. Additionally, it is important to gather information from multiple sources such as parents, teachers, and other individuals familiar with the individual in question. This will provide the most comprehensive picture of the individual’s language processing abilities.

The professional conducting the assessment should also review any past medical records that may be available. This will help them determine if there are any other conditions that could be contributing to the difficulties with language processing.

Once the assessment is complete, the results can then be used to determine if the individual meets the criteria for a diagnosis of LPD. If they do meet the criteria, then the next step would be to create an individualized treatment plan. The treatment plan should focus on identifying strategies and accommodations that will help the individual better understand and process language.

Therapy of language processing disorder

When it comes to treating Language Processing Disorder, there are several options available. The type of treatment chosen will depend on the severity of the disorder and the individual’s age, health history, and lifestyle.

For younger children, speech-language therapy is often recommended to improve communication skills. Therapy may focus on areas such as grammar, word choice, and articulation. It may also include activities like listening to stories, playing games, and practicing conversations. This can help the child to better understand language and improve their ability to express themselves.

For older children and adults, cognitive behavioral therapy (CBT) may be recommended. CBT can help individuals recognize their thought patterns and challenge distorted beliefs about their language abilities. This can provide them with coping strategies to manage their condition. Additionally, medications may be prescribed to manage symptoms like anxiety or depression that can accompany LPD.

Finally, there are some strategies that individuals can use at home to manage their LPD. This includes developing good study habits, using visual aids and diagrams to support understanding, and using mnemonic devices to help remember information. With a combination of professional treatment and these strategies, individuals with LPD can make significant improvements in their language processing abilities.

Therapy options

  1. Speech therapy: Speech therapy can help individuals with LPD improve their language skills and develop strategies for managing language-related challenges.
  2. Occupational therapy: Occupational therapy can help individuals with LPD improve their ability to process and respond appropriately to sensory stimuli, which may be related to their language processing challenges.
  3. Cognitive behavioral therapy (CBT): CBT is a type of therapy that helps individuals with LPD develop coping strategies and change negative thought patterns that may be contributing to their challenges.
  4. Medications: In some cases, medications may be used to help manage specific symptoms of LPD, such as anxiety or hyperactivity.
Resources
  • m B-R, SeoH-S, Ku J-M, et al. Silibinininhibits the production of pro-inflammatory cytokines through inhibition of NF-κB signaling pathway in HMC-1 human mast-cells. Inflammation. Research. 2013;62(11):941-950. doi:10.1007/s00011-013-0640-1.
  • ChapowalA. PetasitesStudy Group. Randomisedcontrolled trial of butterbur and cetirizine for treating seasonal allergic rhinitis. BMJ 2002;324:144-6.
  • Hayes, N. A. and Foreman, J. C. The activity of compounds extracted from feverfew on histamine release from rat mast-cells. J Pharm Pharmacol1987;39(6):466-470
  • Hsieh et al. Baicalein inhibits IL-1ß- and TNF-a-induced inflammatory cytokine production from human mast-cells via regulation of the NF-?B pathway. ClinMolAllergy. 5: 5. 2007.
  • TheoharidesTC, Patra P, Boucher W, et al. Chondroitin sulphateinhibits connective tissue mast-cells. British Journal of Pharmacology. 2000;131(6):1039-1049. doi:10.1038/ sj.bjp.0703672.
  • Ro JY, Lee BC, Kim JY, et al. Inhibitory mechanism of aloe single component (alprogen) on mediator release in guinea pig lung mast-cells activated with specific antigenantibodyreactions. J PharmacolExpTher. 2000;292:114–121. 73.
  • https://www.ncbi.nlm.nih.gov/pubmed/24477254
  • https://www.ncbi.nlm.nih.gov/pubmed/28458279
  • https://www.ncbi.nlm.nih.gov/pubmed/9421440
  • https://www.ncbi.nlm.nih.gov/pubmed/10344773
  • https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4315779/
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