Benefits of neuroplasticity

Benefits of neuroplasticity

Benefit of neuroplasticity

Neuroplasticity can be a powerful tool for restoring normal brain functions, as well as for treating a range of neurological and mental health issues. So neuroplasticity can be beneficial not only in the case of brain injury but autism as well.

Benefits of Neuroplasticity

One of the main benefits of neuroplasticity is its ability to help us recover from physical and mental injuries. For example, neuroplasticity has been used to help stroke victims regain lost motor and sensory functions. Neuroplasticity can also be used to treat neurodegenerative diseases like Parkinson’s, as well as mental illnesses such as depression and anxiety. Neuroplasticity can be used to rewire neural pathways in order to make them stronger, more efficient, and more resilient.

Neuroplasticity can also be used to improve cognitive abilities such as memory, attention, and learning. It has been used to help people with learning disabilities, including dyslexia, by allowing them to make better use of their existing neural pathways. By strengthening existing connections and creating new ones, neuroplasticity can help people learn new skills more quickly.

Neuroplasticity can be used to help us stay mentally fit and healthy throughout our lives. By stimulating our brains in different ways – for example, by learning new things or practicing mindfulness – we can keep our minds sharp and resilient. Neuroplasticity gives us the ability to adapt to changes in our environment or lifestyle, and this can help us stay mentally fit and healthy into old age.

The Limitations of Neuroplasticity

Neuroplasticity is an incredibly powerful and effective tool for healing the brain, but it does have its limitations. For one, neuroplasticity doesn’t work as quickly or efficiently as some people think. It’s important to remember that neuroplasticity can take time to fully manifest in the brain, and depending on the condition, it may not ever fully cure the issue at hand.

Additionally, neuroplasticity isn’t always a permanent fix. The brain is constantly changing and adapting, which means that neuroplasticity may be helpful in the short-term but not necessarily the long-term. It is also possible that changes made through neuroplasticity can become unstable over time, leading to the original problem returning.

Finally, neuroplasticity has its limits in terms of what it can treat. Neuroplasticity is largely focused on healing physical brain issues, such as certain types of brain injuries, rather than mental health issues like depression and anxiety.

Overall, neuroplasticity is a useful tool for healing the brain, but it is important to be aware of its limitations. With proper awareness and care, neuroplasticity can be used effectively to treat a variety of conditions.

Related articles:

Brain plasticity exercises Part1

Can the brain be cured? – What is neuroplasticity?

Resources
  • m B-R, SeoH-S, Ku J-M, et al. Silibinininhibits the production of pro-inflammatory cytokines through inhibition of NF-κB signaling pathway in HMC-1 human mast-cells. Inflammation. Research. 2013;62(11):941-950. doi:10.1007/s00011-013-0640-1.
  • ChapowalA. PetasitesStudy Group. Randomisedcontrolled trial of butterbur and cetirizine for treating seasonal allergic rhinitis. BMJ 2002;324:144-6.
  • Hayes, N. A. and Foreman, J. C. The activity of compounds extracted from feverfew on histamine release from rat mast-cells. J Pharm Pharmacol1987;39(6):466-470
  • Hsieh et al. Baicalein inhibits IL-1ß- and TNF-a-induced inflammatory cytokine production from human mast-cells via regulation of the NF-?B pathway. ClinMolAllergy. 5: 5. 2007.
  • TheoharidesTC, Patra P, Boucher W, et al. Chondroitin sulphateinhibits connective tissue mast-cells. British Journal of Pharmacology. 2000;131(6):1039-1049. doi:10.1038/ sj.bjp.0703672.
  • Ro JY, Lee BC, Kim JY, et al. Inhibitory mechanism of aloe single component (alprogen) on mediator release in guinea pig lung mast-cells activated with specific antigenantibodyreactions. J PharmacolExpTher. 2000;292:114–121. 73.
  • https://www.ncbi.nlm.nih.gov/pubmed/24477254
  • https://www.ncbi.nlm.nih.gov/pubmed/28458279
  • https://www.ncbi.nlm.nih.gov/pubmed/9421440
  • https://www.ncbi.nlm.nih.gov/pubmed/10344773
  • https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4315779/
Can the brain be cured? – What is neuroplasticity?

Can the brain be cured? – What is neuroplasticity?

what is neuroplasticity of the brain

Have you ever wondered if the brain can be “cured”? This is a question that has been debated for centuries, but recent research into the science of neuroplasticity may provide the answer. Neuroplasticity is the idea that the brain can change and adapt throughout our lives, and that these changes can be beneficial to our overall mental health and wellbeing. In this blog post, we will explore what neuroplasticity is, how it works, and whether it can be used to “cure” the brain.

The brain has the ability to change and adapt, a phenomenon known as neuroplasticity. Neuroplasticity refers to the brain’s ability to reorganize itself by forming new connections between brain cells (neurons) and by altering the strength of existing connections. This process allows the brain to adapt and learn new skills, as well as to compensate for injuries or diseases.

Electrical impulses travel through neurons to carry information

A neurotransmitter is released when the body receives it. A neurotransmitter is released when the body receives it. Released neurotransmitters bind to receptors through the synaptic cleft and create new synapses post-synaptic neurons fire electrical signals that cause the brain to fire as a result. The synapse can modify itself according to how strong it is. A neuron’s ability to alter its shape is called neuroplasticity. There is a reason why our brains can constantly learn new information even without us realizing it is a seemingly overburdened system.
Neuroplasticity refers to how a neuron’s brain-cell synapses grow or decrease in strength depending on the use or lack thereof.

Neuroplasticity, sometimes referred to as brain plasticity which is the capability of the brain to form new connections between neurons. Neuroplasticity is an adaptive feature of the brain that enables it to adjust its function in response to changes in the environment or within the body. It allows the brain to modify its structure and circuitry, responding to learning, experience, and injury.
The concept of neuroplasticity is not new; scientists have known about it since the late 19th century. However, it wasn’t until recently that neuroscientists began to understand the full extent of its power and potential.
Neuroplasticity occurs when the brain adapts to external stimuli or internal needs. It involves the modification of existing neural pathways and the formation of new ones. This process allows for a more efficient and effective information organization, leading to improved mental performance and the potential for improved memory, language, and cognition.
Neuroplasticity can occur at any age, and it plays a key role in memory, learning, and the ability to adapt to new situations. Research has also shown that neuroplasticity can be enhanced through certain activities, such as learning new skills, practicing meditation, and engaging in physical exercise.
While neuroplasticity offers the potential for the brain to adapt and regenerate, it is not always possible to fully “cure” certain brain conditions or diseases. In some cases, neuroplasticity may be able to help compensate for damage or dysfunction in the brain, but it is not always possible to fully restore lost function.

The Three Main Types of Neuroplasticity

There are three main types of neuroplasticity: Hebbian plasticity, activity-dependent plasticity, and homeostatic plasticity.

Hebbian plasticity is the type of neuroplasticity where neurons that fire together, wire together. This type of plasticity is based on the idea that when two neurons are activated simultaneously, their connection is strengthened. This is why it is so important to practice new skills or behaviors in order for them to become ingrained in your brain.

Activity-dependent plasticity occurs when neural connections are strengthened or weakened based on experience and activity. This type of plasticity occurs when an individual engages in activities that require concentration and focus. For example, if someone spends a great deal of time practicing a musical instrument, their neural connections associated with playing that instrument will become stronger over time.

Homeostatic plasticity is the type of neuroplasticity that helps regulate the firing rate of neurons. This type of plasticity allows the brain to adjust its responses to different stimuli by fine-tuning the connections between neurons. For example, when someone experiences chronic stress, their brains may increase their sensitivity to stressful stimuli in order to protect itself from further harm. Homeostatic plasticity helps the brain maintain a balance between different states.

How Does Neuroplasticity Work in the Brain?

The first step in the process is for the brain to recognize something new. This could be a new idea, concept, or experience. Once the brain is aware of this new stimulus, it begins to form pathways and connections between different parts of the brain.

It can form these connections in several different ways. For example, the brain may increase communication between two different areas or strengthen an existing connection. It can also weaken connections that are no longer needed or even create entirely new ones.

These changes don’t happen overnight, though. Neuroplasticity is a long-term process, so it takes time for the brain to adjust to the new stimulus and make these changes permanent. The end result is a more efficient and effective brain that can handle complex tasks with ease.

Ultimately, neuroplasticity is an amazing process that helps us learn and adapt to our environment. By understanding how it works, we can develop strategies and techniques to optimize our mental performance and ultimately get the most out of life.

In the case of autism, it is worth taking advantage of the possibility of neuroplasticity, as it can greatly contribute to the development of learning abilities, cognitive abilities, and memory. In our next article, we list tasks that promote neuroplasticity

Related articles:

Brain plasticity exercises Part1

Benefits of Neuroplasticity

Resources
  • m B-R, SeoH-S, Ku J-M, et al. Silibinininhibits the production of pro-inflammatory cytokines through inhibition of NF-κB signaling pathway in HMC-1 human mast-cells. Inflammation. Research. 2013;62(11):941-950. doi:10.1007/s00011-013-0640-1.
  • ChapowalA. PetasitesStudy Group. Randomisedcontrolled trial of butterbur and cetirizine for treating seasonal allergic rhinitis. BMJ 2002;324:144-6.
  • Hayes, N. A. and Foreman, J. C. The activity of compounds extracted from feverfew on histamine release from rat mast-cells. J Pharm Pharmacol1987;39(6):466-470
  • Hsieh et al. Baicalein inhibits IL-1ß- and TNF-a-induced inflammatory cytokine production from human mast-cells via regulation of the NF-?B pathway. ClinMolAllergy. 5: 5. 2007.
  • TheoharidesTC, Patra P, Boucher W, et al. Chondroitin sulphateinhibits connective tissue mast-cells. British Journal of Pharmacology. 2000;131(6):1039-1049. doi:10.1038/ sj.bjp.0703672.
  • Ro JY, Lee BC, Kim JY, et al. Inhibitory mechanism of aloe single component (alprogen) on mediator release in guinea pig lung mast-cells activated with specific antigenantibodyreactions. J PharmacolExpTher. 2000;292:114–121. 73.
  • https://www.ncbi.nlm.nih.gov/pubmed/24477254
  • https://www.ncbi.nlm.nih.gov/pubmed/28458279
  • https://www.ncbi.nlm.nih.gov/pubmed/9421440
  • https://www.ncbi.nlm.nih.gov/pubmed/10344773
  • https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4315779/
Childhood Apraxia of Speech-Symptoms, treatments, exercises

Childhood Apraxia of Speech-Symptoms, treatments, exercises

auditory processing disorder

Childhood apraxia of speech (CAS) is a neurological disorder that affects an individual’s ability to produce speech sounds accurately and fluently. CAS is caused by difficulty planning and coordinating the muscle movements needed for speech, and is not caused by weakness or paralysis of the muscles used for speech.

Symptoms of CAS may include:

  1. Difficulty producing sounds: Children with CAS may have difficulty producing certain sounds or may not be able to say words or sounds correctly.
  2. Difficulty with the rhythm and flow of speech: Children with CAS may have difficulty with the rhythm and flow of speech, and may have a choppy or halting speech pattern.
  3. Difficulty with language skills: Children with CAS may have difficulty with language skills, such as understanding and using words and sentences.

Treatment of CAS

CAS can affect children of any age and can range in severity. Treatment for CAS typically involves speech therapy, which can help children improve their speech production skills and develop more effective communication strategies. In some cases, medications may be used to help manage specific symptoms of CAS, such as anxiety or hyperactivity. It is important to note that treatment for CAS is typically tailored to the individual’s specific needs and may involve a combination of therapies and interventions. A healthcare professional, such as a speech therapist or psychologist, can help develop an appropriate treatment plan.

Treatment for childhood apraxia of speech (CAS) typically involves speech therapy, which can help children improve their speech production skills and develop more effective communication strategies. The goals of speech therapy for CAS may include:

  1. Improving speech sound production: Speech therapy can help children with CAS learn to produce speech sounds more accurately and fluently. This may involve activities and exercises to help improve muscle control and coordination.
  2. Improving language skills: Speech therapy can also help children with CAS improve their language skills, such as understanding and using words and sentences.
  3. Developing compensatory strategies: Speech therapy can help children with CAS develop compensatory strategies, such as using gestures or visual aids, to help them communicate more effectively.

Here are a few exercises that may be beneficial for a child with Childhood Apraxia of Speech (CAS):

  1. Articulation exercises: Children with CAS often struggle with the physical movements needed to produce speech sounds correctly. Exercises that focus on the muscles of the face and mouth, such as blowing bubbles, blowing through a straw, or blowing up balloons, can help to improve the child’s ability to coordinate the movement of their speech muscles.
  2. Repetition exercises: Children with CAS may benefit from repeating words and phrases to improve their ability to coordinate the movements needed for speech. This can be done by repeating simple words or phrases, such as “mom” or “I want juice,” in isolation or in short phrases or sentences.
  3. Imitation exercises: Children with CAS may benefit from imitating the speech of others. The child can watch or listen to a model speak and then imitate what they hear. This can help to improve the child’s ability to coordinate the movements needed for speech.
  4. Auditory bombardment: The child listen to different words and phrases repeatedly, which exposes them to a wide range of sounds and words, which helps them in their development of speech sounds.
  5. Tactile cueing: Children with CAS may benefit from having a tactile cue to remind them of the correct position of the articulators. This can be done by placing an object such as a popsicle stick in the child’s mouth to remind them of the correct position for speech sounds.
  6. Motor planning exercises: Children with CAS struggle with planning and coordinating the muscle movements required for speech, so exercises that target this area can be helpful. this can be done by asking child to move their body in specific ways while they are saying a word or phrase, such as making big arm movements while saying the word “big” or touching their nose while saying “nose.”
Resources
  • m B-R, SeoH-S, Ku J-M, et al. Silibinininhibits the production of pro-inflammatory cytokines through inhibition of NF-κB signaling pathway in HMC-1 human mast-cells. Inflammation. Research. 2013;62(11):941-950. doi:10.1007/s00011-013-0640-1.
  • ChapowalA. PetasitesStudy Group. Randomisedcontrolled trial of butterbur and cetirizine for treating seasonal allergic rhinitis. BMJ 2002;324:144-6.
  • Hayes, N. A. and Foreman, J. C. The activity of compounds extracted from feverfew on histamine release from rat mast-cells. J Pharm Pharmacol1987;39(6):466-470
  • Hsieh et al. Baicalein inhibits IL-1ß- and TNF-a-induced inflammatory cytokine production from human mast-cells via regulation of the NF-?B pathway. ClinMolAllergy. 5: 5. 2007.
  • TheoharidesTC, Patra P, Boucher W, et al. Chondroitin sulphateinhibits connective tissue mast-cells. British Journal of Pharmacology. 2000;131(6):1039-1049. doi:10.1038/ sj.bjp.0703672.
  • Ro JY, Lee BC, Kim JY, et al. Inhibitory mechanism of aloe single component (alprogen) on mediator release in guinea pig lung mast-cells activated with specific antigenantibodyreactions. J PharmacolExpTher. 2000;292:114–121. 73.
  • https://www.ncbi.nlm.nih.gov/pubmed/24477254
  • https://www.ncbi.nlm.nih.gov/pubmed/28458279
  • https://www.ncbi.nlm.nih.gov/pubmed/9421440
  • https://www.ncbi.nlm.nih.gov/pubmed/10344773
  • https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4315779/
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